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Why the Heart Fails and How the Body Compensates

1Cardiac Output and the Determinants of Pump Performance2What Heart Failure Is and Why the Pump Fails3The Frank-Starling Response: Using Preload to Defend Output4Sympathetic Activation: Raising Rate and Contractility5The Renin-Angiotensin-Aldosterone System: Defending Volume and Pressure6Cardiac Remodeling: When Compensation Becomes the Disease
What Heart Failure Is and Why the Pump Fails

How Common Diseases Break the Pump

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Look at the comparison table. Each row shows how a disease changes the heart's structure and function. Myocardial infarction kills muscle cells, so contractility drops and the ventricle dilates — that is systolic failure. Hypertension forces the ventricle to push against high pressure, so it thickens and becomes stiff — that is diastolic failure. Valvular disease can go either way: stenosis creates a pressure load like hypertension, while regurgitation creates a volume load that dilates the ventricle and eventually weakens it. Cardiomyopathy is the clearest example: dilated cardiomyopathy is systolic by definition, and hypertrophic cardiomyopathy is diastolic by definition. The pattern is consistent: the structural change tells you which mechanism is failing.
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Etiology → Mechanism → Failure Type

Myocardial infarction

  • Loss of contractile myocardium
  • Reduced contractility
  • Systolic failure (reduced EF)

Hypertension

  • Chronic pressure overload
  • Concentric hypertrophy, stiff ventricle
  • Diastolic failure (preserved EF)

Valvular disease

  • Pressure or volume overload
  • Stenosis → hypertrophy; regurgitation → dilation
  • Stenosis → diastolic; regurgitation → systolic

Cardiomyopathy

  • Primary myocardial disease
  • Dilated → weak contraction; hypertrophic → impaired filling
  • Dilated → systolic; hypertrophic → diastolic

Why the distinction is not academic

A patient with a reduced ejection fraction and a dilated ventricle has systolic failure; a patient with a normal ejection fraction, a thick-walled ventricle, and dyspnea on exertion has diastolic failure. The two require different management strategies, and the physical exam findings — displaced apex beat in systolic failure versus a sustained but non-displaced impulse in diastolic failure — reflect the underlying geometry. Recognizing the mechanism also predicts which complications are likely: systolic failure tends toward progressive dilation and arrhythmia, while diastolic failure tends toward pulmonary congestion with exertion.

References

  1. [1]Heart Failure: Pathophysiology and Etiologyncbi.nlm.nih.gov
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