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When the Immune System Fails: Mechanisms of Immune Dysfunction

1Normal Immune Defense as a Layered System2Barrier and Innate Failure: When the First Lines Collapse3B-Cell and Antibody Failure4T-Cell and Thymic Failure5Regulatory Failure: Autoimmunity and Allergy
T-Cell and Thymic Failure

What Losing Helper and Cytotoxic T Cells Does to the Body

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The comparison on this page is the key. CD4 cells see peptide on MHC class two, which is displayed mainly by professional presenting cells, and their job is to instruct others: help B cells make antibody, activate macrophages to kill what they have ingested, and license dendritic cells. CD8 cells see peptide on MHC class one, which is on nearly every nucleated cell, and their job is to kill that cell. Now read the two columns together. When CD4 help is lost, macrophages are not activated, so organisms like mycobacteria survive inside them; B cells get weak help, so antibody is poor even though the B cells are fine; and cytotoxic priming suffers too. That is why CD4 loss is the most devastating form. When CD8 killing is lost, infected cells are not destroyed and viruses keep spreading. The right-hand column shows the contrast with humoral failure: encapsulated bacteria dominate there, whereas here you see viruses, fungi, protozoa, and intracellular bacteria. These organisms are not especially virulent; they exploit the specific gap left by missing T-cell function.
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Two subsets, two jobs

CD4 helper T cells recognize peptide on MHC class II, which is displayed mainly by professional antigen-presenting cells. When activated, they secrete cytokines and express surface molecules that instruct other cells: they help B cells produce antibody, they activate macrophages to kill ingested organisms, and they license dendritic cells. CD8 cytotoxic T cells recognize peptide on MHC class I, which is displayed by nearly every nucleated cell, and kill the cell displaying it. Because MHC class I presents peptides from the cell's own cytosol, CD8 cells are the defense against pathogens that live inside cells.

Loss of CD4 help disables several arms at once

Without CD4 help, macrophages that have ingested organisms such as mycobacteria are not activated and the organisms survive inside them. B cells receive weak or no help, so antibody responses are poor even though the B cells are normal. Cytotoxic T cells are also less efficiently primed. A single CD4 defect therefore produces a broad collapse rather than one narrow failure, which is why CD4 loss is the most devastating form of cellular immunodeficiency.

Loss of CD8 killing permits unchecked replication

When cytotoxic T cells are absent, virus-infected cells are not destroyed. Viruses continue to replicate and spread from cell to cell, and the infections that follow are persistent and progressive rather than self-limited. The same applies to intracellular bacteria and protozoa that escape into the cytosol.

Cellular versus humoral failure

Cellular (T-cell) failure

  • Viruses, including persistent and progressive infections
  • Fungi such as Candida and Pneumocystis
  • Protozoa such as Toxoplasma
  • Intracellular bacteria such as mycobacteria
  • Live attenuated vaccines can cause disseminated disease

Humoral (B-cell) failure

  • Encapsulated bacteria: Streptococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis
  • Recurrent sinopulmonary infection
  • Enteroviral meningoencephalitis
  • Live attenuated vaccines are generally tolerated

Why these are called opportunistic

The organisms that dominate cellular immunodeficiency are ones a healthy immune system controls easily. They are not intrinsically highly virulent; they take advantage of the specific gap left by missing T-cell function. This is why the infection pattern itself is diagnostic evidence about which layer has failed.

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